Desonide topical steroids

I have had a rash & pimples on my face since I was a teenager, I am now 34 and am from Africa. I have been using a product called Bio Claire which worked very well the past 10 years but I stopped using it about a month ago because of concerns over an ingredient called BHT in it. I now have an itchy face, with rash around my chin area and over my cheeks and forehead. Sometimes there are small light-coloured patches on my face. My nose and the area around it is free of all blemishes, though it is rather oily. Can you help? I don’t know what to use to cleanse my face or moisturise it or to get rid of the rash.

Hi I’m 25 I have had lip eczema since 12 years old and face eczema since 20 years old. My Lip eczema being moderate and face eczema mild. However this year my face eczema began to flare up. I live in Miami which everyone should know very hot and humid. I should be very oily all year round but I’m just dry, generally in my t-zone. I smoked cigarettes from 16 years old to about 22 years old never thinking anything of it until this year when I experienced a bit of dermatitis and eventually scaling, redness, and itchy eczema. I cannot even wear makeup because it ends up flaking and falling off. For my lip eczema I was given from my dermatologist desonide for the swelling and also a lip balm from Dr. Dan corti balm, which has been like the smallest bit of heaven. Granted the scaling on my lips had not gone away they are always peeling although this has been the best relief I have received thus far. For my face eczema I have been using elidel for about 4 days now twice daily. What I like about this cream is that it’s not a steroid therefore it will not thin out my skin as it is already very thin and flakey. I also use the cetaphil moisturizer in combination with their facial wash when I’m very dry. As for my diet I have always eaten processed foods, spicy foods, and anything that was junk food. About 3 months ago I started dieting and eating fruits, veggies, and salads to reduce any allergic reactions or flare ups. I’m going to post back in a few days to advise everyone of my progress with elidel. God bless you guys!

Topical steroids are available as creams, lotions, gels and ointments; selection of an appropriate product can also provide good moisturization of the skin. The wide spectrum of potencies and bases allows these mediations to be used both effectively and safely while under the care of an experienced physician.

During flares, over-the-counter moisturizing preparations that include a topical corticosteroid (such as clobetasone butyrate and hydrocortisone) are helpful to control inflammation and restore the skin barrier. The intensive use of emollient-based products can reduce the need for topical steroids.

Like other topical corticosteroids, desonide has anti-inflammatory, antipruritic and vasoconstrictive properties. The drug binds to cytosolic glucocorticoid receptors. This complex migrates to the nucleus and binds to genetic elements on the DNA. This activates and represses various genes. However corticosteroids are thought to act by the induction of phospholipase A2 inhibitory proteins, collectively called lipocortins. It is postulated that these proteins control the biosynthesis of potent mediators of inflammation such as prostaglandins and leukotrienes by inhibiting the release of their common precursor arachidonic acid. Arachidonic acid is released from membrane phospholipids by phospholipase A2.

Desonide topical steroids

desonide topical steroids

Like other topical corticosteroids, desonide has anti-inflammatory, antipruritic and vasoconstrictive properties. The drug binds to cytosolic glucocorticoid receptors. This complex migrates to the nucleus and binds to genetic elements on the DNA. This activates and represses various genes. However corticosteroids are thought to act by the induction of phospholipase A2 inhibitory proteins, collectively called lipocortins. It is postulated that these proteins control the biosynthesis of potent mediators of inflammation such as prostaglandins and leukotrienes by inhibiting the release of their common precursor arachidonic acid. Arachidonic acid is released from membrane phospholipids by phospholipase A2.

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