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I disagree with the notion that it “causes” ruptured connective tissues, as there is no real evidence of a direct mechanism, but it is very anti-progestagenic and anti-estrogenic, which can both greatly hinder some of the body’s anti-inflammatory processes and increase the risk of injury. Taking a low dose of deca (say 150-200mg/wk) can effectively counteract this as deca and winstrol, with regards to inflammation , work through the same mechanisms but in opposition of each other. Deca aromatizes somewhat and is also a progestin that activates the progesterone receptors. Winny also binds to the progesterone receptors, but does not activate them, which means it effectively blocks them from being activated.